室内外空气污染对学龄儿童呼吸系统影响的研究

通过对污染区和对照区 2 0 0 0多名学龄儿童呼吸系统症状、疾病患病率比较 ,并运用非条件 Logistic回归分析方法 ,就室内外空气污染对学龄儿童呼吸系统健康的影响 ,进行了研究。结果显示 ,污染区室内外空气污染较严重 ,大气中TSP、SO2 和 NOx 的浓度均超过国家二级标准 ,其儿童呼吸系统症状、疾病患病率明显高于对照区。对儿童呼吸系统发病有显著影响的环境因素是 :大气污染、被动吸烟、家庭燃煤、冬季取暖和室内油烟污染程度。其中咳嗽与以上五个因素有显著的正相关关系 (P<0 .0 5) ;除被动吸烟外 ,支气管炎与其它四个因素呈显著正相关 (P<0 .0 5) ;咳痰与大气污染、被动吸烟、冬季取暖和室内油烟污染程度 ,气喘和呼吸系统疾病住院率与大气污染和室内油烟污染正相关关系显著 (P<0 .0 5)。哮喘与室内外空气污染无显著的统计学联系     

影响儿童呼吸系统病症的混杂因子分析

利用 Logistic回归分析 ,对影响儿童呼吸系统病症的混杂因子进行了分析 ,并在控制了混杂因子后 ,对比数比值(OR)进行了比较 ,发现双亲哮喘和双亲气喘、室内烟雾程度和家长做饭时眼部受刺激频率等因子与儿童呼吸系统健康有比较强的统计关联     

吸烟室内污染对家庭成员呼吸系统健康的影响

应用“家庭健康问卷调查”的方法,对中国兰州,重庆,武汉,广州４城市８所小学中的儿童家庭进行“吸烟室内污染对家庭成员呼吸系统健康的影响”调查,结果表明,吸烟的家庭比不吸烟的家庭室内污染严重,对人体健康有明显影响,其儿童患感冒咳嗽咳痰,支气管等病症的发生率,父亲吸烟的家庭比不吸烟的家庭要高出２倍６倍。     

学龄儿童肺功能的影响因素分析

以武汉市污染区和对照区 50 0多名学龄儿童为研究对象 ,通过问卷调查和为期四年的肺功能连续测定 ,运用逐步回归模型对儿童肺功能的影响因素进行多元分析。结果表明 ,儿童肺功能生长发育水平与机体生理因素 ,尤其是身高的关系最密切。室内外空气污染对儿童肺功能的生长发育有不同程度的抑制作用 ,小气道对各环境因素的影响较大气道敏感 ,女性较男性敏感。影响儿童肺功能最主要的环境因素是大气 TSP,其次为 SO2 、家庭燃煤和 NOx,被动吸烟的影响较轻。TSP与 FEF2 5 %～ 75 % 有着密切的统计学联系 ,是影响小气道功能的主要污染物     

儿童呼吸系统疾病对肺功能影响的探讨

通过 1 995～ 1 996年 3次肺功能测量和家庭健康问卷调查发现 ,患过感冒咳嗽、咳痰、气喘、哮喘和支气管炎的儿童中 FEV1 / FVC、FEV1 /预测值以及 FVC/预测值的异常率比未患过这 5种疾病的儿童都高 ,且前者达到显著性水平。FVC/预测值、FEV1 /预测值有部分疾病异常率达到显著水平。患过呼吸系统疾病 ,且肺功能测量异常的儿童比未患过呼吸系统疾病 ,且肺功能测量正常儿童的 FVC调整预测值下降 3 4 5～ 4 1 8ml,FEV1 调整预测值下降 3 56～ 4 75ml,FEV1 /FVC平均下降 1 4 .2 %～ 1 9.4 %。说明曾患过呼吸系统疾病儿童的肺功能已受到了一定程度的影响     

兰州市空气污染对儿童及其父母呼吸系统健康的影响

叙述了居住环境对儿童和儿童父母亲呼吸系统健康影响的研究结果。揭示了儿童对空气污染所引起的呼吸系统疾病的急性反应较成人更敏感。吸烟合并空气污染更能损害人的健康     

学龄儿童呼吸系统疾病、症状影响因素研究

用非条件 Logistic回归分析方法研究空气污染对学龄儿童呼吸系统疾病、症状的影响。结果表明 ,控制年龄、性别、父亲和母亲受教育水平、住房结构、家庭烹饪燃料、厨房抽风设备、被动吸烟等因素 ,污染区儿童因呼吸系统疾病住院、患支气管炎和出现喘息症状的危险性分别是相对清洁区的 1 .6、1 .5和 1 .2倍 ;不感冒咳嗽或不感冒咳痰的发生与室外空气污染水平的关系均不明显 ;家庭烹饪使用不洁燃料、厨房通风不良、被动吸烟使喘息症状的发生机会增加 (尤其是厨房的通风状况不良 )     

学龄儿童肺功能水平影响因素的研究

用逐步回归分析方法对与室内、外空气污染和机体状况等有关的因素对广州市学龄儿童肺功能 (FVC、FEV1 、PEFR和 FEF2 5～ 75 )水平的影响进行研究。结果提示 ,影响肺功能水平的主要因素是内因 ,身高的影响最明显 ,呼吸系统疾病或症状发生与肺功能水平下降有关 ;室外空气污染使肺功能水平下降 ,SO2 、NOx 和 PM2 .5 的影响明显 ,PM2 .5 的影响明显大于 PM1 0 和 TSP;居室或厨房通风不良、家庭烹饪用不洁燃料对儿童肺功能的生长不利 ;被动吸烟对女性儿童肺功能有不利影响。对各种影响因素 ,PEFR和 FEF2 5～ 75 比 FVC和 FEV1 更为敏感     

2017-2019年扬州市大气污染物与儿童呼吸系统门诊量的时间序列分析

收集扬州市2017—2019年大气污染物细颗粒物(PM_(2.5))、二氧化硫(SO_2)、二氧化氮(NO_2)和臭氧(O_3)的日均质量浓度、气象因素(日均温度、相对湿度)以及每日儿童呼吸系统门诊量数据,运用广义相加模型(GAM)进行单污染物和多污染物分析。结果表明,单污染物分析中,大气污染物的质量浓度累积滞后效应均大于单独滞后效应。ρ(PM_(2.5))、ρ(SO_2)和ρ(NO_2)对儿童呼吸系统门诊量影响效应均在累积滞后7 d(lag 07)达到最大值,ρ(O_3)在累积滞后6 d(lag 06)达到最大值。表现为ρ(PM_(2.5))、ρ(SO_2)和ρ(NO_2)在lag 07时,每升高10μg/m~3,儿童呼吸系统门诊量超额危险度分别为0.720%[95%置信区间(95%CI):0.346%～1.095%]、6.955%(95%CI:5.197%～8.743%)和2.133%(95%CI:1.516%～2.754%);ρ(O_3)在lag 06时,每升高10μg/m~3,儿童呼吸系统门诊量超额危险度为1.160%(95%CI:0.873%～1.447%)。多污染物分析中,当引入所有污染物(SO_2+NO_2+O_3)时,PM_(2.5)对儿童呼吸系统门诊量风险影响消失。大气污染物浓度的升高会增加儿童呼吸系统疾病的发生风险,并且其影响存在一定的滞后效应,有必要开展有针对性的大气污染物与儿童呼吸系统疾病的预警研究,保护易感人群,合理分配卫生资源。     

周边烟雾对石家庄市环境质量的影响机理分析

利用极轨卫星监测、环境监测和实时气象观测资料,针对2002年10月石家庄市出现的严重烟雾污染个例进行了分析,对影响石家庄市环境质量的烟雾污染源区和烟雾扩散输送的范围、强度进行了实时监测;对烟雾污染的机理进行了初步探讨,结果表明:(1)华北中南部持续的逆温层阻挡了烟雾的垂直湍流扩散,使得烟雾在水平方向上长距离输送;(2)河北南部地面较强偏南风及低空流场为烟雾输送提供了动力条件;(3)地面及低空风的辐合及太行山地形影响形成的局地环流使得烟雾在石家庄区域汇集;(4)本地不利扩散的气象条件使得烟雾停滞。     

Indoor radon measurement and effective dose assessment of 150 apartments in Mashhad,Iran

Environmental monitoring and indoor radon measurement are important for public health, to estimate the cancer risk of respiratory system and, if necessary, to suggest proper methods that reduce indoor radon level. In this research, indoor radon concentration in the air has been measured in 150 apartments in Mashhad city. The result demonstrates about 94.7% of apartments have radon concentration less than 100 Bq/m³, taken by WHO as the action level, and 5.3% have the concentration higher than this level. As well as, annual radon dose has been assessed using the equation for annual effective dose calculation introduced by United Nations Scientific Committee on the Effects of Atomic Radiation.     

Indoor air in schools and lung function of Austrian school children

The Children's Environment and Health Action Plan for Europe (CEHAPE) of WHO focuses (inter alia) on improving indoor environments where children spend most of their time. At present, only little is known about air pollution in schools and its effect on the lung function of school children. Our project was set up as an Austrian contribution to CEHAPE. In a cross-sectional approach, differences in indoor pollution in nine elementary all-day schools were assessed and 34 of these pollutants were analyzed for a relationship with respiratory health determined by spirometry using a linear regression model. Overall 596 children (aged 6-10 years) were eligible for the study. Spirometry was performed in 433 children. Socio-economic status, area of living (urban/rural), and smoking at home were included in the model as potential confounders with school-related average concentration of air pollutants as the variable of primary interest. A negative association with flow volumes (MEF(75)) was found for formaldehyde in air samples, benzylbutylphthalate and the sum of polybrominated diphenylethers in school dust. FVC and FEV(1) were negatively associated with ethylbenzene and xylenes in air samples and tris(1,3-dichlor-2-propyl)-phosphate on particulates. Although, in general, the quality of school indoor air was not worse than that reported for homes, effects on the respiratory health of children cannot be excluded. A multi-faceted strategy to improve the school environment is needed.     

The interaction between physical and social-psychological factors in indoor environmental health

Indoor environmental health is now recognized as an important factor in preventing respiratory health problems in the United States. It is also a concern in Canada due to the amount of time that Canadians spend indoors because of cold weather and the potential for increased time indoors during the summer if theclimate warms. The negative health effects are often labeled assick building syndrome, but diagnosing a building or itsoccupants as sick is complicated by the variety of symptoms, thepresence of chronic versus acute symptoms and social andpsychological (socio-psychological) factors that may reduce theeffectiveness of an engineering solution. As a case study, thecontribution of various factors to indoor environmental health,in three buildings at the University of Toronto, was examinedusing five different methods. The results indicate that theinhabitants of the buildings consider features other than airquality in considering building health such as design,maintenance, funding cuts and socio-psychological factors.     

Elevated concentrations of endotoxin in indoor air due to cigarette smoking

Exposure to environmental tobacco smoke (ETS) is an important worldwide public health issue. The present study demonstrates that cigarette smoke can be a major source of endotoxin (lipopolysaccharide, LPS) in indoor environments. Gas-chromatography/mass-spectrometry was used to determine 3-hydroxy fatty acids as markers of endotoxin in air-borne house dust in homes of smokers and non-smokers. Air concentrations of endotoxin were 4-63 times higher in rooms of smoking students than in identical rooms of non-smoking students. The fact that cigarette smoke contains large amounts of endotoxin may partly explain the high prevalence of respiratory disorders among smokers and may also draw attention to a hitherto neglected risk factor of ETS.     

Safe as houses? Indoor air pollution and health

Indoor air pollution has long been the Cinderella of the environmental world: left at home, out of sight and out of mind. But as our knowledge of indoor pollution grows, policy-makers are coming to realise that improving indoor environments can deliver big gains for public health. The new front line on air quality will be on our own doorsteps.     

Human exposure, health hazards, and environmental regulations

United States environmental regulations, intended to protect human health, generally fail to address major sources of pollutants that endanger human health. These sources are surprisingly close to us and within our control, such as consumer products and building materials that we use within our homes, workplaces, schools, and other indoor environments. Even though these indoor sources account for nearly 90% of our pollutant exposure, they are virtually unregulated by existing laws. Even pollutant levels found in typical homes, if found outdoors, would often violate federal environmental standards. This article examines the importance of human exposure as a way to understand and reduce effects of pollutants on human health. Results from exposure studies challenge traditional thinking about pollutant hazards, and reveal deficiencies in our patchwork of laws. And results from epidemiological studies, showing increases in exposure-related diseases, underscore the need for new protections. Because we cannot rely solely on regulations to protect us, and because health effects from exposures can develop insidiously, greater efforts are needed to reduce and prevent significant exposures before they occur. Recommendations include the development and use of safer alternatives to common products, public education on ways to reduce exposure, systematic monitoring of human exposure to pollutants, and a precautionary approach in decision-making.     

兰州市空气污染对儿童肺功能影响的前瞻性研究

从不同的角度阐述了室内外空气污染对儿童肺功能 ,特别是小气道功能的影响及其影响程度的前瞻性研究结果。并通过多因素、多指标的综合分析 ,揭示了一些影响儿童肺功能的主要因素。指出了从保护人体健康的角度来说 ,好的环境空气质量和良好的室内空气质量同样重要