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191.
Pieterse AH Kettunen M Diouf S Ndao I Sarr K Tarvainen A Kloff S Hellsten S 《Ambio》2003,32(7):458-462
The invasion of Salvinia molesta in the Lower Senegal River Delta in Mauritania and Senegal in 1999 posed a serious threat to the socioeconomic conditions of the local people as well as to wetland biodiversity. Eventually, an effective biological control of S. molesta was obtained by means of the weevil Cyrtobagous salviniae, which was introduced in the river in Senegal and Mauritania in May 2000 and in Senegal in April 2001. In October 2001, it became apparent that the weevils were doing a magnificent job. The color of the plants was turning from green to dark-brown or black, and subsequently the plants started to sink to the bottom. Detailed monitoring of the dispersal of C. salviniae in November-December 2001 confirmed the visual observations of the outcome of biological control. In April 2002, it could be concluded that S. molesta was no longer a problem in the Senegal River. 相似文献
192.
Jenkins TF Hewitt AD Grant CL Thiboutot S Ampleman G Walsh ME Ranney TA Ramsey CA Palazzo AJ Pennington JC 《Chemosphere》2006,63(8):1280-1290
Environmental investigations have been conducted at 23 military firing ranges in the United States and Canada. The specific training facilities most frequently evaluated were hand grenade, antitank rocket, and artillery ranges. Energetic compounds (explosives and propellants) were determined and linked to the type of munition used and the major mechanisms of deposition. 相似文献
193.
Warmer does not have to mean sicker: temperature and predators can jointly drive timing of epidemics
Ecologists and epidemiologists worry that global warming will increase disease prevalence. These fears arise because several direct and indirect mechanisms link warming to disease, and because parasite outbreaks are increasing in many taxa. However, this outcome is not a foregone conclusion, as physiological and community-interaction-based mechanisms may inhibit epidemics at warmer temperatures. Here, we explore this thermal-community-ecology-based mechanism, centering on fish predators that selectively prey upon Daphnia infected with a fungal parasite. We used an interplay between a simple model built around this system's biology and laboratory experiments designed to parameterize the model. Through this data-model interaction, we found that a given density of predators can inhibit epidemics as temperatures rise when thermal physiology of the predator scales more steeply than that of the host. This case is met in our fish-Daphnia-fungus system. Furthermore, the combination of steeply scaling parasite physiology and predation-induced mortality can inhibit epidemics at lower temperatures. This effect may terminate fungal epidemics of Daphnia as lakes cool in autumn. Thus, predation and physiology could constrain epidemics to intermediate temperatures (a pattern that we see in our system). More generally, these results accentuate the possibility that warmer temperatures might actually enhance predator control of parasites. 相似文献