环境噪声监测与分析

在大量的资料进行了整理分析的基础上，叙述了近几年重庆市渝中区环境噪声控制所取得的进展，并提出进一步改善声环境质量的一些建议。     

活性碳纤维处理含酚废水的研究

采用活性碳纤维处理苯酚模拟废水，通过静态和动态吸附研究，测定了吸附等温线动态穿透曲线，并研究了溶液的pH值、吸附平衡时间对处理效果的影响。结果表明，活性碳纤维对苯酚的吸 附容量为275.1mg/g，吸附速率快，当溶液pH＞8时，吸附效率下降，吸附时间存在最佳值。吸附饱和的活性碳纤维用10％的氢氧化钠溶液再生，重复使用3次，吸附效率无明显变化。     

采用门限自回归模型预测环境空气质量

应用汤家豪博士提出的门限自回归模型，以上海市环境空气质量时间序列监测数据和相应的气象数据，建立门限自回归大气污染预测模型，对上海市环境空气质量进行预测预报。结果表明：门限自回归模型计算较为简便且便于计算机自动建模。显著性检验表明门限自回归预报方程高度显著，在实际应用中，采用最新的数据建模和采用分站建立模型的方法可以使预测精度进一步提高。     

大学校园的生态足迹探究

生态足迹分析法是评价绿色校园建设水平较好的指标之一,能定量揭示学校的生态状况,本文结合黑龙江科技学院的校园状况,简要介绍生态足迹成分法的原理和计算分析方法.研究结果表明,黑龙江科技学院2005年总生态足迹为18410.53 hm2,即需要18410.53 hm2生态生产性土地来支持学校的各类消费以及吸纳废弃物;人均生态足迹为0.94 hm2/人,与全国人均生态足迹1.5 hm2相比较,学校的生态状况比较乐观.     

昆明红壤环境下酸沉降对二种树木生长的影响

将昆明红壤人工酸化后进行树木生长实验。发现昆明红壤的酸缓冲能力非常大。在 12 0meqH /Lsoil的酸添加处理区也没有发现树木生长的显著下降。根据实际情况判断 ,认为没有必要继续加大酸添加强度进行实验     

西昆仑造山带花岗岩研究新进展

西昆仑造山带包括西昆仑山、喀喇昆仑山和帕米尔高原部分地区,是青藏高原有机组成部分.它位于古亚洲构造域与特提斯构造域结合部位,大地构造位置特殊,研究意义重要.区内岩浆活动强烈,花岗岩广泛出露.已有许多学者对该区花岗岩作过研究,但以往的研究基本沿中巴公路和新藏公路进行,并侧重于年代学方面,还没有从花岗岩岩石系列和成因类型(前人主要报道过钙碱性系列和Ⅰ型花岗岩)以及构造-岩浆演化等方面作深入研究.本次工作深入到西昆仑造山带腹地,对区内不同时期有代表性的22个花岗岩体进行了地质、地球化学综合研究,取得了如下重要进展和创新认识:     

CFB锅炉SNCR烟气脱硝空预器堵塞原因分析及处理

选择性非催化还原(SNCR)法是目前CFB锅炉常用的烟气脱硝技术。对一台锅炉空气预热器堵塞的原因进行了研究,对堵塞物成分、喷氨、燃煤硫分、空气预热器吹灰等进行了分析。通过煤种调整、喷氨改造、合理投运空预器吹灰等手段,成功解决了SNCR脱硝导致空预器堵塞的问题。     

Intranasal administration of tetrabromobisphenol A bis(2-hydroxyethyl ether) induces neurobehavioral changes in neonatal Sprague Dawley rats

Tetrabromobisphenol A (TBBPA) and its derivatives are now being highly concerned due to their emerging environmental occurrence and deleterious effects on non-target organisms. Considering the potential neurotoxicity of TBBPA derivatives which has been demonstrated in vitro, what could happen in vivo is worthy of being studied. Tetrabromobisphenol A bis(2-hydroxyethyl ether) (TBBPA-BHEE), a representative TBBPA derivative, was selected for a 21-day exposure experiment on neonatal Sprague Dawley (SD) rats through intranasal administration. The neurobehavioral, histopathological changes, and differentially expressed genes based on RNA microarray were investigated to evaluate the neurological effects of this chemical. The results indicated that TBBPA-BHEE exposure significantly compromised the motor co-ordination performance and the locomotor activities (p < 0.05). The neurobehavioral phenotype could be attributed to the obvious histopathological changes in both cerebrum and cerebellum, such as neural cell swelling, microglial activation and proliferation. A total of 911 genes were up-regulated, whereas 433 genes were down-regulated. Gene set enrichment analysis showed multiple signaling pathways, including ubiquitin-mediated proteolysis and wingless-int (Wnt) signaling pathway etc. were involved due to TBBPA-BHEE exposure. The gene ontology enrichment analysis showed the basic cellular function and the neurological processes like synaptic transmission were influenced. The toxicological effects of TBBPA-BHEE observed in this study suggested the potential neuronal threaten from unintended exposure, which would be of great value in the biosafety evaluation of TBBPA derivatives.