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651.
On East Asian Sand and Duststorms and Associated Significant Dustfall Observed from January to May 2001 总被引:4,自引:0,他引:4
Monitoring of dust pollution at the western shore of Tae-ahn Peninsula (TAP) and in the Chongju area of central Korea was carried out from January to May 2001. It was found that in Koreathere were 9 cases of sand and duststorms (DS) and 16 associatedsignificant dustfall (SD) days. Observed maximum concentrations of DS and SD coming from NW China and Mongolia were in the rangeof 300–920 for TSP, 200–690 for PM10 and 100–170 g m-3 for PM2.5.Satellite measurements clearly showed the formation and subsequent movement of DS to the Korean Peninsula and onward to the Korea East Sea, Japan and the Gulf of Alaska. According to satellite image analysis of dust clouds there were abundant coarse particles, measuring in size of 11.0 m. Medium-sizedparticles measuring in the range of 3.5–7.0 were also prevalent,while fine particles of less than 2.0 m were less distinctive in reflectivity. Measured values of PM2.5 were alsorelatively low with SD events.The measured average pH values of dusty precipitation associatedwith DS were 7.24. Alkaline precipitation can play a `temporary'role in the neutralization of acidified soil until the subsequentevent of acidic rain. The new selection criteria of SD days from PM2.5 at 85 g m-3, PM10 190 g m-3 and TSP 250 g m-3 are recommended on dust pollution occurring from the invasion of a DS elsewhere. 相似文献
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654.
为了保证数字遥测地震台网的地震监测和速报质量,在<地震观测技术规范>规定各遥测台站要每天(甚宽带要每月)定时进行脉冲标定,并每天检查脉冲标定的幅度和周期变化,当发现脉冲标定波形的周期发生明显变化或脉冲幅度变化>5%时,应及时检查修复.以前天津台网定时进行脉冲标定,但脉冲标定周期和幅度都是通过值班人员人工检测,并记录在值班日志上的方法进行检查的.现在我们编写了<脉冲标定自动检测软件>,软件每天能自动完成检测并将结果通过对话框的方式显示在微机上,使这项手工费时费力的检测工作变成抄表式操作. 相似文献
655.
检出限的大小主要取决于仪器的灵敏度、稳定操作和操作技能等因素。通过精密度偏性试验、由空白值批内标准偏差估算检出限,得出检出限综合估计值。 相似文献
656.
Gabriella Restagno Marina Ferrone Carlo Doriguzzi Laura Palmucci Tiziana Mongini Angelo Carbonara 《黑龙江环境通报》1995,15(7):672-674
The sister of a child affected by Duchenne muscular dystrophy (DMD) was referred for genetic counselling to assess the risk of her being a carrier. Her brother had died 15 years previously at the age of 8. There were no other affected males in the family. There were no methods for DNA investigation at the time of the child's death and the family had never been studied for linkage with polymorphic probes on the chromosomal region Xp21. The only tissue from which an assessment of the risk could be made by DNA linkage analysis was two of the child's deciduous teeth that the parents had kept. DNA was extracted using a protocol described for the recovery of ancient DNA from museum specimens and archaeological finds. Multiplex amplification did not reveal deletions in 19 exons spanning the hot-spot regions for deletions within the dystrophin gene in Xp21. Linkage analysis using three highly polymorphic microsatellites demonstrated that the sister had not received the X chromosome borne by her brother. These results show that DNA extracted from teeth is a reliable source for molecular diagnosis. 相似文献
657.
Prenatal diagnosis was requested by a family carrying a 3 base-pair insertion in the dihydropteridine reductase (DHPR) coding region. A chorionic villus sample was obtained and fetal DNA was isolated directly from this. Diagnosis was performed by a polymerase chain reaction (PCR)-based technique, with a simple electrophoretic assay for the insertion. The fetus was found to be heterozygous for the insertion. This is the first time that prenatal diagnosis of DHPR deficiency has been performed by direct detection of the mutation. 相似文献
658.
Professor V. S. Baranov V. N. Gorbunova O. V. Malysheva O. V. Artemyeva T. K. Kascheeva O. V. Evgrafov A. V. Polyakov V. M. Lebedev T. V. Kuznetzova S. N. Shlykova A. V. Mikhailov V. G. Vakharlovsky 《黑龙江环境通报》1993,13(5):323-333
Of 126 families referred for counselling of Duchenne muscular dystrophy (DMD), DNA analysis has been suggested to 119 families with at least one affected child or with an affected close male relative of the woman at risk of being a DMD carrier. A large proportion (about 80 per cent) of the families were represented by sporadic cases (only one affected individual). By means of multiplex polymerase chain reactions with different sets of oligoprimers providing amplification of 10–11 different exons, altogether 49 dystrophin gene deletions were identified (41 per cent). Eighteen deletions clustered in the 5′ ‘hot spot’ region of DMD cDNA and 36 in the distal half of the central rod domain around exons 43–53. An unusually high frequency (18 per cent) of deletions involving exons 17–19 was discovered. Large deletions extending through both ‘hot spot’ regions and thus occupying over 30–40 exons were recorded in five cases (10 per cent). Seventy-six of 94 families were found to be informative by RFLP analysis for intragenic or extragenetic DNA probes. Carrier status was ascertained in 20 and rejected in 32 female relatives in 40 DMD families. Eight DMD-affected fetuses were diagnosed prenatally by direct deletion testing or by RFLP analysis. Feasible interpopulation variations in the dystrophin gene deletion pattern are discussed. The prospects for more effective prenatal diagnosis and carrier detection in high-risk DMD families in Russia are briefly outlined. 相似文献
659.
Dr. Naomichi Matsumoto Nakamichi Saito Naoki Harada Kiyoji Tanaka Norio Niikawa 《黑龙江环境通报》1995,15(7):675-677
DNA-based prenatal carrier detection of group A xeroderma pigmentosum (XP-A) is reported. Chorionic villus sampling was done at the tenth gestational week in a pregnant woman whose first child suffers from XP-A. Genomic DNAs from the villi, proband, and parents were PCR (polymerase chain reaction)-amplified using three sets of primers, because the PCR and a subsequent enzyme digestion with HphI, AlwNI, or MseI may detect the three most frequent mutations of the XP-A complementing gene (XPAC) in Japanese XP-A patients. The results showed that the proband is a homozygote and that the parents and fetus are heterozygotes for a base substitution at the 3′ acceptor site of intron 3 of XPAC, indicating that the fetus is a healthy carrier of XP-A. This is the first case of prenatal carrier detection of the disorder. 相似文献
660.