Simultaneous removal of ammonium and phosphate by zeolite synthesized from coal fly ash as influenced by acid treatment

Zeolite synthesized from fly ash （ZFA） without modification is not efficient for the purification of NH4＋ and phosphate at low concentrations that occur in real effluents, despite the high potential removal capacity. To develop an effective technique to enhance the removal efficiency of ammonium and phosphate at low concentrations, ZFA was modified with acid treatment and the simultaneous removal of ammonium and phosphate in a wide range of concentration was investigated. It was seen that when compared with untreated ZFA, only the treatment by 0.01 mol/L of H2SO4 significantly improved the removal efficiency of ammonium at low initial concentrations. The behavior was well explained by the pH effect. Treatment by more concentrated H2SO4 led to the deterioration of the ZFA structure and a decrease in the cation exchange capacity. Treatment by 0.01 mol/L H2SO4 improved the removal efficiency of phosphate by ZFA at all initial P concentrations, while the treatment by concentrated H2SO4 （≥0.9 mol/L） resulted in a limited maximum phosphate immobilization capacity （PIC）. It was concluded that through a previous mild acid treatment （e.g. 0.01 mol/L of H2SO4）, ZFA can be used in the simultaneous removal of NH4＋ and P at low concentrations simulating real effluent.     

叠层橡胶支座串联隔震体系抗火性能研究

对叠层橡胶隔震支座在高温作用下的温度场进行数值模拟,利用小模型试件进行温度传导及温度场分布试验。在叠层橡胶隔震支座经历420℃的高温作用后,将隔震支座模型试件与钢筋混凝土柱串联,并对串联隔震体系的力学性能进行压剪往复试验。结果表明,叠层橡胶隔震支座在高温作用下的性能退化相当明显,承载状态下的橡胶隔震支座在稍高的温度作用下就开始发生畸变。这表明在实际工程中对叠层橡胶隔震支座采取防火保护措施对保证隔震结构承载力安全非常重要。     

Toxicity of depleted uranium on isolated liver mitochondria: a revised mechanistic vision for justification of clinical complication of depleted uranium (DU) on liver

Depleted uranium (DU) is widely used in military anti-armor weapons. Recent evidence suggested that oxidative stress and mitochondrial dysfunction may contribute to DU-induced toxicity. However, the underlying mechanisms of DU toxicity in mitochondria are not well understood. In this study, liver mitochondria were obtained from Wistar rats treated with DU in the form of uranyl acetate (UA) (0.5, 1 or 2 mg/kg i.p.) using differential centrifugation. For in vitro experiments, control rat liver mitochondria were incubated with different concentrations of UA (50, 100 or 200 μM) for 1 hr. Mitochondrial reactive oxygen species (ROS) production, collapse of mitochondrial membrane potential, and mitochondrial swelling were examined by flow cytometry. Mitochondrial sources of ROS formation were determined using specific substrates and inhibitors. Extent of lipid peroxidation (LPO) and glutathione (GSH) oxidation, and also complex II and IV activities were detected via spectroscopy. Further, the concentration of ATP and ATP/ADP ratio was measured using luciferase enzyme and release of cytochrome c from mitochondria which was detected by ELISA kit. UA induced succinate-supported mitochondrial ROS production, elevated LPO levels, GSH oxidation, and mitochondrial complex II inhibition. UA also induced mitochondrial permeability transition and increase in cytochrome c release which subsequently disturbed oxidative phosphorylation and reduced the mitochondrial ATP concentration. Data suggest that mitochondrial oxidative stress and uncoupling of oxidative phosphorylation may play key roles in DU-induced hepatic toxicity.     

Toxicity of cigarette smoke on isolated lung,heart, and brain mitochondria: induction of oxidative stress and cytochrome c release

Cigarette smoking is one of the main risk factors for premature human death which is associated with a variety of respiratory and vascular diseases, and cancer due to exposure to hundreds of toxicants. Rat mitochondria were obtained by differential ultracentrifugation and incubated with different concentrations (1%, 10%, or 100%) of standardized cigarette smoke extract (CSE). Our results showed that CSE induced a rise in mitochondrial reactive oxygen species (ROS) formation, lipid peroxidation, and mitochondrial membrane potential (MMP) collapse before mitochondrial swelling ensued in isolated pulmonary mitochondria. Disturbance in oxidative phosphorylation was also confirmed by decrease in ATP concentration in the CSE-treated mitochondria. In addition, collapse of MMP and mitochondrial swelling produced release of cytochrome c via outer membrane rupture or mitochondrial permeability transition (MPT) pore opening. Our results suggested that CSE-induced toxicity in lung tissue is the result of disruptive effect on mitochondrial respiratory chain that leads to ROS formation, lipid peroxidation, MMP decline, and cytochrome c expulsion which results in apoptosis signaling and cell loss.     

Assessment of the role of thymol in combating chromium (VI)-induced oxidative stress in isolated rat erythrocytes in vitro

Thymol, the main phenolic compound in Thymus vulgaris, has been shown to have various biological effects. The main objective of this study was to investigate the efficacy of thymol on counteracting hexavalent chromium-induced oxidative damage in rat erythrocytes in vitro. The radical scavenging activity of thymol was examined using the 1, 1-diphenyl-2-picrylhydrazyl assay. Erythrocytes resistance to oxidative damage, lipid peroxidation, osmotic pressure, hemolysis as well as morphological alterations were evaluated in the presence of 2.5 µg thymol mL^?1 with or without 5 µmol hexavalent chromium mL^?1 of the incubation media. Results from the 1,1-diphenyl-2-picrylhydrazyl assay denoted good radical scavenging activity of thymol. Thymol caused a significant increase in superoxide dismutase and catalase activities and reduced glutathione content in erythrocytes intoxicated with hexavalent chromium. In contrast, the presence of thymol resulted in markedly less-elevated malondialdehyde levels, hemolysis, and destabilization of erythrocytes exposed to hexavalent chromium. Microscopically, thymol markedly reduced hexavalent chromium-induced morphological alterations in rat red blood cells. Conclusively, thymol counteracted hexavalent chromium-induced oxidative damage in rat erythrocytes.     

孤岛工作面长度对下伏大巷稳定性影响研究

以开滦矿区某矿8#孤岛煤柱回采为工程背景,针对孤岛工作面长度对底板垂直应力的分布影响及下伏大巷围岩稳定性问题展开研究。采用理论分析和数值模拟相结合的手段,建立了孤岛工作面在倾向方向上的底板受力模型,采用半无限体空间理论,推导出底板垂直应力计算公式,将该孤岛工作面实际地质参数带入公式,得出了孤岛工作面长度与底板应力之间的函数曲线。研究表明:当该孤岛工作面长60 m时,对下方-480 m水平大巷产生的垂直应力最小。通过FLAC 3D数值模拟方法验证了8#孤岛工作面合理尺寸（60 m)比实际孤岛工作面长度（100 m)对下方水平大巷的采动影响小,便于维护。     

基于Ventsim的南山煤矿孤岛工作面均压通风方案研究*

孤岛工作面掘进期间,采用在原有采空区内掘进进风巷、回风巷、切眼的方案以降低工作面冲击地压风险等级。为了降低此布置方案产生的采空区漏风对工作面的影响,设计了6种均压通风方案,使用Ventsim三维通风仿真软件对工作面实施6种方案后通风系统和风压分布状态进行模拟。通过对模拟结果比较,确定以局部通风机吸风量400 m3/min（方案3）为最优设计方案,并根据方案3对该工作面实施均压通风。对该工作面通风系统实测结果分析表明,局部通风机出口风量为346.8 m3/min,进、回风巷及工作面的通风阻力分别为22.8,29和15.2 Pa,验证了该方案的有效性及可靠性,通风系统风量能保证工作面正常开采,且漏风量较小。最后,对工作面采煤过程中以及推进到停采线时均压通风系统进行了稳定性分析,并提出了管理措施。     

Investigation of Building - Influenced Atmospheric Dispersion Using a Dual Source Technique

Dispersion of atmospheric contaminants in the vicinity of an isolated cubical model building was investigated in the field. A dual source/receptor technique was used in the experiments, which was proved to be very useful for the investigation of pollution dispersion. This experimental technique involved the simultaneous release of two different tracer gases from two different point sources, and the deployment of a FID (Flame Ionization Detector) co-located with a UVIC®(Ultra-Violet Ion Collector) detector. Both mean concentrations and concentration fluctuation statistics were examined. In this paper concentration fluctuation statistics are presented. The effect of the upwind source location on intermittency values and on the cumulative density function (cdf) is examined. The exact location of a source placed upwind of an obstacle has a very significant and complex effect on both mean concentrations and concentration fluctuations. As the lateral or vertical displacement between the two sources is increased, cross-correlation values between data taken simultaneously by two co-located detectors decrease rapidly.     

高层隔震结构非线性地震响应分析及设计方法研究

高层隔震结构的分析理论和设计方法是目前隔震技术向高层建筑推广的2个关键理论问题。本文以高烈度区宿迁市已经竣工的高层隔震建筑阳光大厦为工程实例,对高层隔震结构的非线性地震响应分析方法进行了研究,包括隔震支座竖向不同拉压刚度的模拟、各种类型的隔震支座水平力学特性的准确模拟,以及不同地震动输入方式和输入角度对隔震支座受拉情况的影响分析等;在此基础上,对隔震层的设计方法进行了分析,提出了隔震层设计的基本原则,进而给出了控制隔震层设计的基本指标,包括隔震支座的长期面压、极值面压、隔震层偏心率等;最后,对高层隔震结构的相关构造要求进行了必要的说明。     

基础隔震结构抗震可靠度简化分析方法

选用胡聿贤平稳地震地面运动模型作为基础隔震结构的随机地震动输入,采用Bouc-Wen模型描述隔震结构的层间滞变位移,将滞变体系动力特性矩阵随机等效线性化,并建立等价线性状态方程.引入左右特征向量系,对振动微分方程进行解耦,推导了基础隔震结构随机地震响应的统计矩解析解.采用变形失效准则,定义了上部结构和隔震层的功能状态极...