Effects of acute γ-irradiation on the aquatic microbial microcosm in comparison with chemicals

Effects of acute γ-irradiation were investigated in the aquatic microcosm consisting of green algae (Chlorella sp. and Scenedesmus sp.) and a blue–green alga (Tolypothrix sp.) as producers; an oligochaete (Aeolosoma hemprichi), rotifers (Lecane sp. and Philodina sp.) and a ciliate protozoan (Cyclidium glaucoma) as consumers; and more than four species of bacteria as decomposers. At 100 Gy, populations were not affected in any taxa. At 500–5000 Gy, one or three taxa died out and populations of two or three taxa decreased over time, while that of Tolypothrix sp. increased. This Tolypothrix sp. increase was likely an indirect effect due to interspecies interactions. The principal response curve analysis revealed that the main trend of the effects was a dose-dependent population decrease. For a better understanding of radiation risks in aquatic microbial communities, effect doses of γ-rays compared with copper, herbicides and detergents were evaluated using the radiochemoecological conceptual model and the effect index for microcosm.     

Radionuclides in cigarettes may lead to carcinogenesis via p16 inactivation

It is widely accepted that tobacco smoke is responsible for the vast majority of lung cancers worldwide. There are many known and suspected carcinogens present in cigarette smoke, including α-emitting radioisotopes. Epidemiologic studies have shown that increased lung cancer risk is associated with exposure to ionizing radiation, and it is estimated that the majority of smoking-induced lung cancers may be at least partly attributable to the inhaled and deposited radiation dose from radioisotopes in the cigarette smoke itself. Recent research shows that silencing of the tumor suppressor gene p16^INK4a (p16) by promoter methylation plays a role in smoking-related lung cancer. Inactivation of p16 has also been associated with lung cancer incidence in radiation-exposed workers, suggesting that radionuclides in cigarette smoke may be acting with other compounds to cause smoking-induced lung cancer. We evaluated the mechanism of ionizing radiation as an accepted cause of lung cancer in terms of its dose from tobacco smoke and silencing of p16. Because both radiation and cigarette smoking are associated with inactivation of p16, and p16 inactivation has been shown to play a major role in carcinogenesis, ionizing radiation from cigarette smoke likely plays a role in lung cancer risk. How large a role it plays, relative to chemical carcinogens and other modes of action, remains to be elucidated.     

移动通信基站电磁辐射时空分布及衰减特征

根据理论计算公式和实测数据,研究基站电磁辐射水平、垂直方向的衰减趋势,分析影响基站电磁辐射时空分布的各种因素,认为基站电磁辐射实际影响范围要远小于安全防护距离。     

首钢孟家冲尾矿坝渗流稳定分析

针对孟家冲尾矿库坝面出现的渗水现象,对坝体的渗流以及稳定进行计算分析。计算结果表明,由于堆积坝的不断升高,导致坝体浸润线抬高,安全稳定系数降低,从而将造成坝体失稳,出现溃坝事故。为消除隐患,提出采用辐射井排渗的设计方案,并对该方案的可行性进行计算分析。     

乌鲁木齐市电磁辐射污染的调查及分析

报道了乌鲁木剂市电磁辐射污染的调查结果,阐明了乌鲁木齐市电磁污染的现状和分析状况,并依据调查结果对各类污染源的电磁泄漏进行了分析讨论。     

北京核与辐射恐怖事件风险分析

采用风险分类-要素分析-风险排序的思路,对北京市核与辐射恐怖事件风险进行分析。根据国际上1998—2006年该类恐怖事件的实例,从事件所涉及的材料、场所角度将核与辐射恐怖事件分为6大类。结合北京市的实际情况进行涉核恐怖事件的要素分析,将北京市各类场所划分为4类敏感区并与5类涉核恐怖事件结合起来,得到北京市核与辐射恐怖事件风险构成图。通过风险分析,笔者认为北京市发生核与辐射恐怖事件风险最大的场所为核研究机构,最容易发生的核与辐射恐怖事件类型为涉及放射源的恐怖事件,结合分析结果对从阻止、探测、响应3个层次上提出了奥运核安保工作的反核恐工作建议。     

一种移动通信基站发射天线非轴向功率密度预测模式

以移动通信基站轴向功率密度计算公式为基础,根据非轴向方向上实际监测点位和轴向方向的相对位置,对轴向功率密度计算公式参数进行系列修正,从而建立一种简单适用的移动通信基站非轴向方向上功率密度预测模式.     

北京市某高层建筑高空电磁辐射环境影响分析

选取北京市某代表性高层居民楼顶平台作为高空电磁辐射环境水平监测点进行综合场强和选频监测,评价总体电磁辐射环境水平,并分析监测区域电磁辐射能量的来源。结果表明：该高空监测区域的电磁辐射环境总体水平低于国家规定的公众照射导出限值,但高于地面水平。监测区域主要受30 km 外的中波台影响,其次是9 km 外的中央电视塔。影响最大的是639 kHz频率的中波信号,占100 kHz～300 MHz总场强的95％以上,其次是1．28 MHz频率的中波信号。提出,开展敏感建筑物规划建设时,应考虑中波的传播特性和对环境的影响。     

泉州市移动通信基站电磁辐射环境影响分析

对泉州市移动通信GSM基站和TD—SCDMA基站电磁辐射状况进行监测与分析。通过理论计算预测基站电磁辐射安全距离,对典型敏感目标、典型基站进行电磁辐射的监测,结果与理论预测情况相符。选取302个GSM基站和306个TD—SCDMA基站进行电磁辐射的监测,结果表明,各基站电磁辐射满足单个项目管理低于目标限值0．08W／m2的标准。分析表明,基站在正常运行时,对周围的电磁辐射环境影响能满足国家相应标准的要求。     

徐州市辐射环境质量现状分析

按照江苏省辐射环境质量监测实施方案,徐州市对大气、水、土壤进行了布点监测,通过分析2007—2011年辐射环境监测数据,得出结论为:徐州市辐射环境整体质量为良好,且相对稳定,在未来几年内,仍将保持良好的状态。