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Giovanni Nigro Renato La Torre Eleonora Sali Maura Auteri Manuela Mazzocco Luca Maranghi Erich Cosmi 《黑龙江环境通报》2002,22(7):558-561
Cytomegalovirus (CMV) is the leading infectious cause of prenatal neurological damage, which is particularly severe when primary maternal infection occurs during the first 16 weeks of gestation, at the time of organ development and neuronal migration. Vascular involvement has been suggested to be among thepossible pathogenic mechanisms of virus-induced pathology, in addition to direct viral effects. We report on a fetus with cerebral CMV infection, which had intraventricular haemorrhage, together with oligohydramnios and hyperechogenic bowel, following maternal primary CMV infection. Copyright © 2002 John Wiley & Sons, Ltd. 相似文献
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Angela Carletti Giulia Gandolfi Colleoni Antonella Perolo Giuliana Simonazzi Tullio Ghi Nicola Rizzo Gianluigi Pilu 《黑龙江环境通报》2009,29(4):389-395
Although no precise figures are available, many congenital brain lesions arise from intrauterine disruption, frequently due to obstetric complications. The most common entities include intracranial hemorrhage, ischemic lesions, thrombosis of venous vessels and infections. Accurate prenatal diagnosis is possible in many of these cases. However, the findings may be subtle, particularly in the early stage of the disruptive process. Identification of these conditions requires therefore specific expertise, the combination of fetal neurosonography and magnetic resonance, and frequently there is a need for serial examinations. Targeted diagnostic imaging should be offered to obstetric patients with conditions predisposing to prenatal cerebral insults. Copyright © 2009 John Wiley & Sons, Ltd. 相似文献
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Professor Umberto Nicolini Alessandra Kustermann Beatrice Tassis Roberto Fogliani Andrea Galimberti Elena Percivalle Maria Grazia Revello Giuseppe Gerna 《黑龙江环境通报》1994,14(10):903-906
Fifteen fetuses at risk of congenital human cytomegalovirus (HCMV) infection underwent prenatal diagnosis at 16–30 weeks' gestation by a combination of amniocentesis and fetal blood sampling. HCMV was isolated from the amniotic fluid in six patients, but HCMV-specific IgM was detected in only three of them. Two of the nine neonates, who were delivered following a negative prenatal diagnosis, had congenital HCMV infection diagnosed by virus isolation in the urine. The interval from infection to prenatal testing was 3 and 4 weeks in the two false-negative cases and ⩾ 7 weeks in the true-positive cases. Although timely testing for HCMV infection allows the option of termination of pregnancy, it may be flawed by false-negative results. 相似文献
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