珠三角SO_2污染区划及来源的定量研究

文章利用珠三角粤港空气质量监测网13个空气质量监测点2009年11月SO2监测浓度,通过聚类分析对珠三角SO2进行污染特征区划,最终选定该时段内3个核心关注区域,分别为广州中心城区、佛山江门片区、珠江口西侧片区。发现佛山江门片区的污染最为严重,其次是珠江口西侧片区。根据一定标准,选定11月23-27日为典型污染过程,11-16日为典型清洁时段。利用MM5-CALPUFF模式对这3个区域,在典型污染过程与清洁时段内的SO2平均浓度进行来源分析,发现在选定的模拟时段内,广州中心城区在污染过程中以本地污染为主(贡献率达62.47%),清洁时段则广州本地污染(49.43%)与东莞输送污染(40.27%)并重;佛山江门片区在污染过程和清洁时段均以外地输送污染(分别为65.06%和68.92%)为主,主要输入源地是广州和东莞;珠江口西侧片区在污染过程和清洁时段均是输送污染与本地污染并重(污染过程输送污染44.10%,本地污染51.83%;清洁时段输送污染50.25%,本地污染41.11%),其中污染过程中,输送污染的主要输入源地是深圳与东莞,清洁时段输送污染比重略大于本地污染,主要输入源地只有东莞。研究结果为控制及治理珠三角SO2源提供科学依据。

SO2 Pollution Regionalization and Pollution Sources Analysis in Pearl River Delta

Using SO2 concentration data measured at thirteen air quality monitoring sites in the Pearl River Delta(PRD) in November 2009,through cluster analysis,the sites were divided into several groups and focuses on three important ones,induding urban areas of Guangzhou(District 1),Foshan-Jiangmen district(District 2) and western coast of Pearl River estuary(District 3) respectively.District 2 is the most badly polluted one and District 3 comes the second.A period from Nov.23 to Nov.27 is selected as pollution-process,and another one from Nov.11 to Nov.16 is selected as clean-process.Then MM5-CALPUFF model is used to analyze the SO2 sources contribution for these three districts in the two processes.Result shows that the SO2 in District 1 mainly originates from the local sources in the pollution-process(the contribution rate 62.47%),but mainly comes from both Guangzhou and Dongguan areas in the clean-process(49.43% and 40.27% respectively).District 2 is polluted by SO2 transported from Guangzhou and Dongguan in both the pollution-process(65.06%) and the clean-process(68.92%).For the District 3,local and external sources make almost the same contributions(44.10% and 51.83% respectively in pollution-process;50.25% and 41.11% respectively in clean-process);the pollutant comes from Shenzhen and Dongguan in the pollution-process,and only from Dongguan in the clean-process.The result could provide an advice to the SO2 controlling.