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排序方式: 共有3630条查询结果,搜索用时 15 毫秒
1.
Zhang Xiuxiu Yi Guiyun Zhang Zhengting Yu Jia Fan Haiyang Li Peng Zeng Huihui Xing Baolin Chen Lunjian Zhang Chuanxiang 《Environmental science and pollution research international》2021,28(12):14671-14680
Environmental Science and Pollution Research - Due to the merits of their high adsorption and convenient separation, magnetic graphene-based composites have become a promising adsorbent in terms of... 相似文献
2.
Liu Shun Jia Xu Xin Long 《Environmental science and pollution research international》2021,28(16):20157-20173
Environmental Science and Pollution Research - The existing literature on the environmental Kuznets curve (EKC) fails to investigate the spatial attribute of the “pollution halo” effect... 相似文献
3.
Long Yingtao Liang Jianjun Xue Yinghao 《Environmental science and pollution research international》2021,28(47):67098-67107
Environmental Science and Pollution Research - A novel composite (nZVI/Pd-AC) was prepared by loading nanoscale zero-valent iron (nZVI) and Pd on activated carbon (AC) electrode under... 相似文献
4.
利用2013年11月武汉市逐日空气质量资料、地面气象观测资料、NCEP/NCAR再分析资料和L波段雷达探空资料,通过WRF模式模拟空气污染生消过程中的局地气象条件变化,探讨特殊地形条件下边界层结构变化和局地环流在污染物生消过程中的作用和影响。结果表明:(1)武汉地区当背景环流场强的时候,由地形引起的局地流场对污染物扩散的影响就弱,反之当背景环流场弱的时候, 地形对流场影响明显:夜间为山风,白天为谷风。夜间山风与偏西北气流及偏东气流在武汉及周边地区辐合,形成气流汇聚带,在武汉地区形成一个反复污染带,即由地形引起的局地流场对污染物扩散的贡献就大;(2)武汉地区发生空气污染时,地面湿度较高,边界层呈上干下湿状态,其特征为暖而干且有偏东小风,这导致污染物不断堆积和重污染过程的形成。 相似文献
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This study investigated the levels, sources and ecological risks of 16 polycyclic aromatic hydrocarbons (PAHs) in two sediment cores that were collected along the Huaxi Reservoir. The spatial distributions and residue levels of the 16 priority PAHs in the sediments from the Huaxi Reservoir were analyzed for their potential ecological risk, source apportionment and contribution to the total PAH residue. The concentration level of the total PAHs (TPAHs) was in the range 1805 ng·g?1 to 20023 ng·g?1 based on dry weight, and the content of PAHs in the Huaxi Reservoir exhibited a gradual upward trend. The PAH congener ratios fluoranthene/(fluoranthene + pyrene) and indeno[1, 2, 3-cd]pyrene/(indeno[1, 2, 3-cd]pyrene + benzo[g, h, i]perylene) were used to identify the source. The main source of the low molecular weight PAHs was wood and coal combustion, whereas the high molecular weight PAHs were primarily from petroleum combustion sources. The results of an ecological risk assessment demonstrated that ACE poses a potential ecological risk, while FLU, NAP, ANT, BaP, DBA, PHEN and PYR can have serious ecological risks. 相似文献
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Luo Xiaofang Dessie Wubliker Wang Meifeng Duns Gregory J. Rong Nianhang Feng Luya Zeng Jianqing Qin Zuodong Tan Yimin 《Journal of Material Cycles and Waste Management》2021,23(2):548-556
Journal of Material Cycles and Waste Management - The traditional Chinese medicinal plant Magnolia officinalis has a wide range of applications; including more than 200 kinds of patented Chinese... 相似文献
9.
Wang Jing Long Zehua Min Wei Hou Zhenan 《Environmental science and pollution research international》2020,27(35):43929-43941
Environmental Science and Pollution Research - Biochar has been widely accepted as a soil amendment to improve nitrogen (N) use efficiency, but the effect of biochar on N transformation metabolic... 相似文献
10.
AbstractThe roles of PM2.5-induced mitochondrial damage and oxidative stress on mast cell degranulation were examined in vitro. Mast cells were treated with suspensions of PM2.5 in Dulbecco’s modified Eagle’s medium at concentrations from 25 to 200?mg/L in the absence or presence of 10?mmol/L N-acetyl-L-cysteine. Biological effects and mitochondrial function were assessed by determining cell viability, β-hexosaminidase release, interleukin-4 secretion, reactive oxygen species generation, adenosine triphosphate production, potential alteration of mitochondrial membrane, and activities of mitochondrial electron transport chain complexes I and III. Exposure of mast cells to PM2.5 induced reduction of adenosine triphosphate production, collapse of mitochondrial membrane potential, and inhibition of the activity of complex III. Co-treatment of mast cells exposed to PM2.5 with N-acetyl-L-cysteine attenuated cytotoxicity and the production of reactive oxygen species, and decreased the release of β-hexosaminidase and interleukin-4. Evidently, PM2.5-induced oxidative stress plays an essential role in mitochondrial toxicity and mast cell activation. 相似文献